Impaired lactate release in dCA1 astrocytes contributed to nociceptive sensitization and comorbid memory deficits in rodents.
Authors
Han S, Jiang B, Ren J, Gao F, Wen J, Zhou T, Wang L, Wei X
Abstract
Memory deficits are a common comorbid disorder in patients suffering from neuropathic pain. The mechanisms underlying the comorbidities remain elusive. We hypothesized that impaired lactate release from dysfunctional astrocytes in dorsal hippocampal CA1 contributed to memory deficits.
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